Communion Of Dreams


You can’t tell a book …

So, a week or so ago I linked to a new review of Communion of Dreams which was very positive overall. But the reviewer made a comment which echoes things some other people have said:

Another item that would likely help get this book moving is a different cover. I understand the imagery now that I’ve read the book, but definitely think it will keep hard-core sci-fi fans from buying a copy (and people do judge books by their covers).

Like I said, every so often a comment to this effect will pop up in a review. And I don’t spend much time thinking about it (and I’m not going to change the cover image at this point), but now and then I wonder just what kind of a cover would appeal to ‘hard-core sci-fi fans’ and still make any kind of sense in relation to the story. Maybe some nice images of Saturn or Titan from the Cassini mission? A depiction of some of the spacecraft (which aren’t described in much detail in the book), or perhaps the Titan Prime space station? Go with a charming post-apocalyptic montage of ruined cities and microphotographs of viruses? To me, none of these would fairly represent the story, and to a certain extent would unnecessarily limit the appeal to only ‘hard-core sci-fi fans’.

But I’m curious what others think. So feel free to post a comment here or over on FB. Over even on Amazon, as a comment on an extant review or in  new review of your own. In a week or so I’ll go through all the various comments I can find, and pick someone to get a jar of my latest hot sauce (or something else if they don’t want that).

Jim Downey

PS: there’s another new short review up on Amazon you might want to take a look at as well.



It’s the End of the Year as we know it…

So, the WordPress Machine informs me that I’ve had a fairly busy year blogging here.

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As I mentioned a while ago, earlier this month I had fallen prey to the nasty bit of cold virus going around.  Turned out that the damn thing was even more stubborn for my wife, who is still struggling with a hacking cough and various other annoying symptoms.  We’ve been keeping a close eye on it, watching for signs of secondary pneumonia, which would call for antibiotic intervention, but I think she’ll get past this on her own.

Which is good, because there really isn’t much we can do to fight a virus. In this sense, medical science is at about the same place in viral treatments as we were in dealing with bacterial infection 70 years ago:

In 1941, a rose killed a policeman.

Albert Alexander, a 43-year-old policeman in Oxford, England, was pruning his roses one fall day when a thorn scratched him at the corner of his mouth. The slight crevice it opened allowed harmless skin bacteria to slip into his body. At first, the scratch grew pink and tender. Over the course of several weeks, it slowly swelled. The bacteria turned from harmless to vicious, proliferating through his flesh. Alexander eventually had to be admitted to Radcliffe Hospital, the bacteria spreading across his face and into his lungs.

Alexander’s doctors tried treating him with sulfa drugs, the only treatment available at the time. The medicine failed, and as the infection worsened, they had to cut out one of his eyes. The bacteria started to infiltrate his bones. Death seemed inevitable.

* * * * * * *

You may not have heard much about it here, but the norovirus is causing all kinds of grief in the UK. Cases are up 83% over last year, and are estimated to have hit over a million people already. In the UK the norovirus is commonly called the “winter vomiting bug” whereas here we tend to call it “stomach flu”.  As miserable as it makes people feel, it’s usually not a life-threatening disease for otherwise healthy people, and the best thing to do is just ride it out.

Of course, public health authorities have taken steps to try and limit the spread of the disease into populations where the virus could be life-threatening, and a lot of hospitals have curtailed or eliminated visiting hours. Furthermore, appeals have been made to the public to not to go see their doctors or go to emergency rooms for routine cases of the norovirus, since there is little that can be done to treat the virus and this just contributes to the spread of the disease.

Still, people get scared when they get sick, even when they know that it is a fairly common bug that’s going around — and one that most people have had before and gotten over just fine. So they tend to swamp available medical services, overwhelming the health care system.

Just think about what would happen if it was a disease which wasn’t known. And one which was killing people so quickly that they’d drop over in the street on the way home from work.

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I’ve been thinking about that a lot, since it is an integral plot point to St. Cybi’s Well.  This isn’t a spoiler, since the advent of the fire-flu is part of the ‘history’ of Communion of Dreams.

But it is something which has had me in a bit of a quandary this fall, as I’ve been working on writing St. Cybi’s Well.

Howso? Well, because I kept going back and forth on making one final decision: where to end the book.

See, I know how the *story* plays out — I’ve had that all sorted since I first worked up the background for Communion of Dreams. But in going to write St. Cybi’s Well, I needed to decide exactly where in the story that book would end. Which is to say, I needed to decide how much, if any, of the onset of the fire-flu would be included. Because I could set everything up and have the book actually finish at the onset of the fire-flu — after all, the reader would know what was about to happen. Why drag the reader through that horror?

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A week or so ago I made my decision, and I’ve been chewing it over since then as I’ve been busy with other things, making sure that I was comfortable with what I have decided, and why. I’m not going to give you the details, but you can safely assume from what I’ve said in this post that at least some of the pandemic will be portrayed.

I decided this not because I have a desire to write about the horror (in spite of what I may have said previously) but rather because it is critical for character development of the main character.

Poor Darnell.

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So, the WordPress Machine informs me that I’ve had a fairly busy year blogging here. 293 posts (this makes 294), which is a faster pace than in some years. Of course, I’ve had a lot of promotional stuff do to with the launch of Communion of Dreams last January and everything to support that through the year, not to mention the Kickstarter for St. Cybi’s Well.

And while I’ve cautioned that I won’t be writing quite as much here on the blog as I’m working on St. Cybi’s Well, well, it does make for a nice change of pace.

So thanks for being along for the ride this year. Together we can see how things go in 2013.

 

Jim Downey

 

 



The other 90% of you.

Your body has something on the order of 10 trillion individual cells. But surprisingly, it has nine or ten times that number of microorganisms which it hosts in some capacity or another, many of which we have co-evolved with and which seem to be critical to our long health. While these microorganisms are typically much smaller than human body cells, in one very real sense, “you” is actually only about 10% “you.”

These microorganisms have a substantial impact on how your body digests food. On whether you can resist various kinds of infection or develop any of a range of auto-immune diseases. Perhaps even on your mood and risk assessment.

Would it therefore be any kind of a surprise at all if doing something to change the “mix” of these microorganisms had an impact on you?

Hell, it’d be a surprise if it didn’t.

Almost all of us know what happens when you have to take a broad-spectrum antibiotic: usually some degree of diarrhea and intestinal discomfort. And in the last decade or two it has become commonplace for people to seek out some variety of probiotics, frequently in the form of live yogurt, as a way to replenish gut flora following antibiotic treatment. I do it myself.

So, extending that idea a bit, researchers are now investigating whether part of the slow-moving plague of obesity can be due to the changes created in the human-hosted microorganisms:

Early use of antibiotics linked to obesity, research finds

The use of antibiotics in young children might lead to a higher risk of obesity, and two new studies, one on mice and one on humans, conclude that changes of the intestinal bacteria caused by antibiotics could be responsible.

Taken together, the New York University researchers conclude that it might be necessary to broaden our concept of the causes of obesity and urge more caution in using antibiotics. Both studies focus on the early age, because that is when obesity begins, the scientists say.

As I’ve noted previously:

In Communion I have a post-pandemic society, one which is recovering from a massive disruption caused by a flu virus which caused rapid death in a large percentage of the population. But the reality of what we’re dealing with might be even more insidious.

More insidious in this case because we have done it to ourselves.

And perhaps not even with direct antibiotic treatment to deal with some kind of life-threatening infection. Consider that it is still a widespread practice to boost livestock weight gain through the use of antibiotics, and that leaves a residue of antibiotics in the meat. If it boosts weight gain in feed animals, why wouldn’t it do the same to us?

I’ve said before that there has been some kind of change to the way our bodies absorb nutrients in the last 40 or 50 years, and that that is behind the global rise in obesity. Previously there were indications that it might be due to some kind of virus. Or an immune response to the germaphobia of the 20th century. But maybe it is more directly our own damned fault, and we’ve traded the ability to defeat infections for a different kind of health risk.

Jim Downey



Details, details.

From the first page of Chapter 17:

“Sorry.” She looked over at him, the dread in her eye replaced by something else. “The 1918 flu was recreated in the early part of this century, as there was a growing concern about Avian flu. The scientists at the time discovered that the prevailing form of Avian Flu, the H5N1 virus, was surprisingly related to the 1918 pandemic virus. Almost identical RNA structure, similar DNA.”

“But you say this one is different.”

“Yeah. Ignis was such a nasty bug because it spread by aerosol, but it also had a very short incubation period, just a couple of days. Then the disease itself was very swift, and victims died within hours of onset. Like it was all time- compressed, hyper-virulent. This is one of the reasons that people thought then, and still debate now, whether it was a weaponized version of Avian flu.”

From yesterday’s All Things Considered:

Anyone and everyone can now look in the journal Science and read about how to make lab-altered bird flu viruses that have been at the center of a controversy that’s raged for months.

But in the eyes of some critics, the details of these experiments are effectively the recipe for a dangerous flu pandemic.

The H5N1 bird flu virus isn’t normally contagious between people, but these mutants most likely are. They were created with the best of intentions by a lab that’s trying to understand how flu viruses might change in the wild and start spreading in humans.

But as word of the experiments got out, scary headlines warned of a “doomsday” virus and a “super-flu.” Scientists, public health officials and security experts debated what to do in closed-door meetings, as others made their arguments in op-eds and at public meetings.

Remember when the “fire-flu” (Latin name: Ignis) was supposed to happen?

Yeah, 2012.

Draw your own conclusions.

Jim Downey




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